I mean destructive to me. Sitting at SS, Threshold or V02 (in whatever percentage that is) But it sort of correlates with this study - Six weeks of a polarized training-intensity distribution leads to greater physiological and performance adaptations than a threshold model in trained cyclists
by Craig M. Neal
Angus M. Hunter
D. Lee Hamilton
Giuseppe DeVito, and
Stuart D. R. Galloway
"The urinary metabolomics data are interesting since a significant model was only observed from pre- to post-training for THR. Of course, it could be argued that the metabolites of interest may reflect dietary influences (caffeine intake, phytochemical intake, protein intake), as has been reported in previous metabolomics and nutrition studies (54, 56) and highlighted in a review by Gibney et al. (18). However, dietary intake was controlled with participants replicating their food intake for 2 days prior to the morning first-pass urine sample collections. Participants did not exercise on the day before collection of the samples, and the THR and POL training interventions were administered in a randomized crossover fashion. Therefore, it would seem that dietary intake would be an unlikely key factor here. Alternatively, the metabolites of interest could collectively suggest differences in cellular metabolic/energy stress induced by the THR training. Greater creatinine excretion would normally reflect greater plasma creatinine degradation, act as a marker of glomerular filtration rate, or mirror lean body mass in 24-h urine collections (15). In morning first-void urine, it likely reflects changes in hydration status or possibly reflects energy availability (55). In the absence of dietary influences, urinary dimethylamine is thought to reflect intermediary metabolism (37), whereas hypoxanthine reflects purine nucleotide degradation, which tends to be acutely lower if high-intensity sprint exercise is not undertaken (50). Urinary 3-methylxanthine can be produced by demethylation of theophylline in the presence of oxidizing radicals (44), and increased urinary excretion of this metabolite could therefore represent greater overall oxidative stress from the THR training period. Changes in hippurate excretion are typically associated with gut microflora (62), and activities of gut microflora may play an important role in energy metabolism and/or immune function of the whole organism (28).
Whereas the metabolomics profile change following THR but not POL cannot be fully explained, it may provide some insight into the overall cellular metabolic/energetic stress experienced with the THR training model. Greater evidence of cellular metabolic/energy stress with the THR model would support the notion of longer recovery times from threshold training sessions or may just reflect the higher training load. Either way, this greater stress was not associated with greater adaptation, which may suggest a maladaptive response to the THR training"
Yep, there are limitations to the study (sample size) and a few what if’s, but I think it can be disseminated and ideas applied.
But I’m a cyclist, not a coach or scientist:mask:
The Polarised concept is a Sprinkling of High intensity to the easy is the way forward. I don’t think (could be wrong) I mentioned staying at LT1 all the time
Oh, sorry if I wasn’t allowed to post that research